They have discovered that mice are closely related to humans genetically...
Mouse or not my friend, were all mamels.
And the technology proven in the above video is that hair follicles by reawakening genes which were once active only in the embryo stage of human development can start all over again.
One of the factors affecting in the slow progress of finding a cure for Stargardt?s is the nature of the gene responsible for this retinal disease. Some people with the gene never develop the disease, while others seem to develop it without the gene. Environmental and unknown factors play a role as well.
In the future, when a patient manifests a genetic defect, doctors hope to be able to correct it and resolve the problems it causes. Gene therapy involves inserting a functioning gene into human cells to correct a genetic error or to introduce a new function to the cells. The surgeon must transplant genes rather than cells, a strategy which miniaturizes the process by a million fold. The surgical tools are not hand-held instruments, but trained viruses. At the 1998 conference of the Association for Research in Vision and Ophthalmology, a report presented just such a technique for retinitis pigmentosa (RP), another retinal degenerative disease. Researchers showed that a virus called Lenti could carry a new gene into mouse photoreceptors and could prevent the photoreceptors from degenerating for six months in a mouse model of recessive RP. That's a long time for a mouse! In another experiment, an adenovirus was used to deliver a gene designed to prevent the expression of an abnormal dominant gene for RP. Thus, it appears that both dominant and recessive forms of RP are amenable to gene therapy -- at least in mice! Before we can begin human trials of gene therapy for retinal degenerations, the long term effects of these viruses, including rejection, must be better understood. Like transplantation, this approach requires delicate subretinal surgery to place the virus near the photoreceptors and the RPE.
